Acute respiratory distress syndrome has a greater incidence among patients in intensive care unit. Acute respiratory distress syndrome leads to impaired respiration. Most patients with acute lung injury often progress into acute respiratory distress syndrome.
Clinical Trials and Research Studies on Acute Respiratory Distress Syndrome
New York (USA), June 27, 2013
Acute Respiratory Distress Syndrome: Clinical Trials and Research Studies
Acute respiratory distress syndrome occurs due to sepsis, mechanical ventilation, smoke inhalation, pneumonia, drug reaction or overdose, major surgery, trauma, shock, and reperfusion pulmonary edema after lung transplantation.
Acute respiratory distress syndrome has a greater incidence among patients in intensive care unit or ICU of a hospital. Most patients with acute lung injury often progress into acute respiratory distress syndrome.
Mechanical ventilation is an important part of acute respiratory distress syndrome treatment. Acute respiratory distress syndrome leads to impaired respiration. Mechanical ventilation eases respiratory muscles and opens your closed airways. Nonetheless, mechanical ventilation is not a foolproof system. It can worsen your acute respiratory distress syndrome situation. Possibility of infection is an inherent aspect of mechanical ventilation. Further, such pressure ventilation alters lung mechanics.
Elevated abdominal pressure is a risk factor for acute respiratory distress syndrome, specifically during mechanical ventilation.
Inflammation as present during sepsis causes endothelial dysfunction, poor drainage of fluid from lungs, and affects fluid discharge from capillaries. Type II pulmonary epithelial cells do not function normally due to reduced surfactant production. Most immune cells lack oxygen and hence increased concentration of oxygen is essential at this stage. Loss of aeration is responsible for important changes in lungs due to inflammation.
Breathing is difficult as pulmonary edema increases thickness of alveolo-capillary space. Oxygen has to traverse greater distance to reach blood and diffuse. This causes problems in gas exchange leading to hypoxia and eventually fibrosis of airspace.
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